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AOR, Antioxidant Synergy, 120 vcaps - Antioxidant Synergy includes the complete "Antioxidant Network," the body's five-member antioxidant defense and recycling system. Most antioxidants lose their protective power once they have quenched a single free radical. These five antioxidants - R(+)-l
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AOR, Antioxidant Synergy, 120 vcaps

AOR Antioxidant Synergy - 120 vcaps
Antioxidant Synergy - 120 vcaps, aor vitamins supplements
AOR, Antioxidant Synergy, 120 vcaps is manufactured by AOR Supplements

Last updated on 12/5/2016

Antioxidant Synergy

  •  Keeps antioxidants in an active state
  •  Protects against cellular damage
  •  Synergistic actions

Antioxidant Synergy provides antioxidants for the maintenance of good health.

NPN Product Code Size Per Capsule Vegetarian
80024549 AOR04010 120 Vegi-Caps 276 mg Vegetarian
Supplement Facts
Serving Size: 4 Capsules
 

R(+)-lipoic acid (sodium salt)* 200 mg
Vitamin E Complex 400 mg
Mixed Tocopherols:  350 mg
RRR-Alpha-Tocopherols 52.8 mg AT
Gamma-Tocopherols 202.4 mg
Delta-Tocopherols 94.8 mg
Mixed Tocotrienols 50 mg
Vitamin C (magnesium ascorbate, ascorbyl palmitate) 420 mg
Coenzyme Q10 30 mg
N-acetylcysteine (NAC) 50 mg
Selenium (selenomethionine) 50 mcg
Resveratrol 5 mg

*Contains 5 mg sodium per capsule. Non-medicinal ingredients: Rosemary extract (5 mg), silicon dioxide. Capsule: hypromellose.

AOR Guarantees: that no ingredients not listed on the label have been added to the product. Contains no wheat, gluten, corn, nuts, dairy, eggs, fish, shellfish or animal byproducts.

Adult Dosage: Take 1 capsule four times daily with food, or as directed by a qualified health care practitioner.

Cautions: Consult a health care practitioner prior to use if you have diabetes or cystinuria, or if you are taking blood pressure medication, blood thinners, nitroglycerin or antibiotics. Consult a health care practitioner for use beyond duration 6 weeks.

Pregnancy/Nursing: Consult a health care practitioner

Source:
Tocopherols: soy. Tocotrienols: palm.

Main Indications:
As reported by literature: • Core antioxidant function

Research

Background Information

Problems with Antioxidants

Did you know that once many antioxidants have quenched a free radical, they actually become mild free radicals themselves? Do not be concerned! This is a natural and quite important part of the oxidation-reduction signaling process. However, when some antioxidants are consumed in excess, they can actually cause more damage than good! The most well studied example of this phenomenon is tocopherol-mediated peroxidation, or TMP, in which vitamin E family members block an incoming free radical from attacking a particle of LDL (“bad”) cholesterol, but the “radicalized” antioxidant then initiates a slower, more insidious pattern of lipid peroxidation. Several studies suggest that TMP may play a devastating role in the long-term development of atherosclerosis. So then yet other antioxidants are required to quench this process. But some antioxidants are more effective in certain locations in the body than others, meaning that you need to take the right antioxidants to halt this vicious cycle of antioxidants turning pro-oxidant! How can this vicious cycle be stopped?

Read More

The One & Only Antioxidant Network

The vicious cycle of oxidation can be prevented through taking advantage of the unique synergistic interactions of an elite antioxidant strike force: the Networking Antioxidants. When taken together, these specific, biologically essential nutrients form a dynamic team of synergistic co-antioxidants. Networking antioxidants can recycle one another from their radicalized forms back into their active, antioxidant forms. By this process of mutual regeneration, networking antioxidants enhance and extend one another’s capacities, working within and around the mitochondria where most of the oxidation occurs. There are exactly five networking antioxidants: R (+)-lipoic acid, the vitamin E complex (including the four tocopherols and four tocotrienols), vitamin C, coenzyme Q10, and glutathione (GSH).

The networking antioxidants have a genuine synergy with one another. The effects of each networking antioxidant support greater functionality of the antioxidant network as a whole. No other antioxidants participate in the interlocking cycles of the antioxidant network. In fact, the ability of other antioxidants to play a protective role in the body depends on having a functional antioxidant network – but not vice-versa.

How Does the Network Function?

First, the original free radical is neutralized by a networking antioxidant. Unfortunately, the result is that the networking antioxidant is degraded into its free radical form. That antioxidant is rejuvenated by another co-antioxidant from the network team. A game of electron donating “hot potato” ensues, which ultimately results in rejuvenation of the last networking antioxidant free radical by dihydrolipoic acid (DHLA, the charged form of R-lipoic acid). And at this point, the “hot potato” game is halted when R-lipoic acid is cycled through the mitochondrial energy-production process.

Network Supporters

A few antioxidants do play a supporting role to networking antioxidants, without fully participating in the antioxidant network recycling system. The best understood of these network-boosters are bioflavonoids and the mineral selenium. Among bioflavonoids, carnosic acid – which is found in the herb rosemary – is especially interesting because of its ability to repeatedly rearrange itself into a “cascade” of new antioxidant “booster” forms before being exhausted. Selenium supports the network by maintaining the body’s supply of two key enzymes: glutathione peroxidase (GSH-Px) and thioredoxin reductase (TrxR). Only very low doses of selenium are needed to maximize the levels and activity of these enzymes. Resveratrol, found in grapes and wine, is a gene-activator in the mitochondria that helps reduce free-radical production in the first place.

Extra Information

The antioxidant network showing the interaction between vitamin E, ubiquiol, vitamin C, glutathione and R-lipoic acid redox cycles. (Packer L, Kraemer K, Rimbach G. “Molecular aspects of lipoic acid in the prevention of diabetes complications.” Nutrition. 2001 Oct; 17(10): 888-95.)

Thiol redox cycles play central roles in the antioxidant defense network. Both glutathione and lipoate redox cycles can be driven by cellular-reducing equivalents to generate their respective reduced forms (GSH and DHLA). The ability of lipoate to increase cell GSH is mediated by the reduction of cystine to the GSH precursor cysteine by dihydrolipoate. LA, lipoate; GSSG, oxidized glutathione; vit, vitamin. (Sen CK & Packer L. “Thiol homeostasis and supplements in physical exercise.” Am J Clin Nutr. 2000 Aug; 72(2 Suppl): 653S-69S.)

Market Trends

Some antioxidants have gained phenomenal reputations and hold a lot of market traction, such as Vitamins C & E and CoQ10. Others like resveratrol have benefited from “fountain-of-youth” hype. While these are noteworthy antioxidants, it may be more beneficial to take a scientifically-proven combination of antioxidants rather than dosing with large amounts of any single one.

AOR Advantage

AOR’s Antioxidant Synergy provides a scientifically-backed combination of antioxidants, not just a mish-mash of the most popular ones on the market.  Get the most out of your antioxidant regime with Antioxidant Synergy!


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