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AOR Omega Natal - 100 softgels


C$ 18.98






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Omega Natal is a fish oil concentrate high in docosahexaenoic acid (DHA), an omega-3 fatty acid found in high concentrations in the brain and retina.  Recent research has demonstrated that DHA can enhance cardiovascular health and also helps support cognitive health/brain function.  Omega Natal is molecular-distilled to remove contaminants such as heavy metals and organochlorines (including PCBs).

100 softgels
AOR04215


SUPPLEMENT FACTS:
Serving Size: 1 softgel


Molecular-Distilled Mixed Fish Oil Concentrate 500 mg

     DHA (Docosahexaenoic Acid 22:6: w 3)

250 mg
     EPA (Eicosapentaenoic Acid 20:5: w 3) 100 mg

Non-medicinal ingredients: glycerol, water, beeswax, ascorbyl palmitate, d-alpha tocopherol, soybean oil. Capsule: gelatin, caramel.

AOR™ guarantees that no ingredients not listed on the label have been added to the product. Contains no wheat, gluten, corn, nuts, dairy, or eggs. 

Suggested Use
Take one to three softgels daily with meals, or as directed by a qualified healthcare practitioner. 

Main Applications
Fetal development
Brain and Heart Health 

Source
Stable (low peroxide), molecular distilled mixed fish oils. 

Cautions
None known 

Pregnancy / Nursing
Safe at recommended dose

*These statements have not been evaluated by the Food and Drug Administration. This product is not intended to diagnose, treat, cure, or prevent any disease.

Omega Natal is a fish oil concentrate high in docosahexaenoic acid (DHA), an omega-3 fatty acid found in high concentrations in the brain and retina. Supplementation with Omega 3 fatty acids during pregnancy and lactation is beneficial for the health of both the child and the mother. Omega-3 fatty acids, and especially docosahexaenoic acid (DHA), are essential for healthy infant development both before and after birth.


The Importance of DHA during Pregnancy and Nursing

Fish lipids, particularly lipids from cold-water fish are high in long chain polyunsaturated fatty acids, especially omega-3 fatty acids, of which DHA (docosahexaenoic acid) is a major constituent.  DHA is a 22-carbon chain containing 6 unsaturated bonds all of which are in the cis configuration.  DHA is an essential part of cellular membranes, specifically in brain and retinal cells - representing roughly 15% of the total fatty acid content of the brain and constituting the predominant fatty acid in the retina. This makes DHA especially important during pregnancy, a period of rapid fetal brain development.  Furthermore, around 80% of fetal DHA accumulation occurs in the last trimester of pregnancy because this is when the fetus builds up adipose tissue. Unfortunately, this leaves preterm babies without the DHA needed for brain maturation and even 40 weeks post-conception, brain DHA levels in preterm infants remain lower.

Because omega-6 and omega-3 share the same metabolic enzymes and compete with each other, modification of the fatty acid content of the diet also changes the fatty acid content of cellular membranes, which in turns affects cellular function and growth.  Breast milk contains higher concentrations of long chain polyunsaturated fatty acids (LCPUFA) than infant formulas.  This may explain why studies have shown that breast-fed infants have higher IQ’s as early as six months after birth, with differences still present at 15 years of age, and also why studies have shown that breast-fed infants have a better visual acuity prior to six months. Animal studies demonstrated that inadequate omega-3 fatty acid supply in the postnatal period affects nerve growth factors, dopamine production and glucose uptake in the brain. Furthermore, brain hypothalamic DHA levels did not recover in animals given alpha-linoleic acid (precursor to omega-3 fatty acids) for 24 weeks after being fed low levels of omega 3.

During gestation, LCPUFA are delivered to the fetus via the placenta.  Human studies have confirmed that higher maternal omega-3 fatty acid consumption may improve cerebral maturation of the newborn and prevent the recurrence of preterm delivery with fish oil supplementation in the last trimester of pregnancy (providing 920 mg DHA and 1.3 g EPA) reducing recurrences from 33% to 21%.Low seafood consumption, which relates to poor omega-3 fatty acid intakes, throughout pregnancy was also shown to be a strong risk factor for preterm delivery and low birth weight. Associations between maternal blood DHA levels and the baby’s sleep patterns were also reported, suggesting that higher maternal blood DHA levels are associated with greater central nervous system maturity in the newborn. Epidemiological data suggests that higher consumption of omega-3 fatty acids throughout pregnancy increases birth weight, head circumference, birth length and the duration of gestation. Animal diets enriched in omega 6 fatty acids and low in omega 3 fatty acids increased blood pressure later on in life.  The International Society for the Study of Fatty Acids and Lipids recommends at least 300 mg DHA per day with a total of at least 650 mg combined DHA and EPA during pregnancy.  These goals could only be reached in the United States through supplementation, a four-fold increase in fish consumption or functional foods.  Furthermore, the concern for an increase in oxidative stress in women given fish oil supplements (unsaturated oils are susceptible to oxidation) has been disproved by Shoji et al.  In their study, pregnant women were given 500 mg DHA and 150 mg EPA fared no worse than women not receiving the supplement.

Another interesting observation was inspired by the notion that omega-6 fatty acids tend to promote inflammation while omega-3 fatty acids exhibit anti-inflammatory activity.  In a recent study, scientists from three American universities looked at the ratio of omega-6 to omega-3 fatty acid in placental tissues of normal and preeclamptic women.  DHA levels were lower in preeclamptic women with a ratio of omega-6 to omega-3 fatty acids roughly twice as high in preeclampsia versus normal pregnancy. The same authors had previously reported lower omega-3 fatty acid concentrations in preeclamptic maternal plasma. In other studies, a 15% increase in the omega-3 to omega-6 ratio reduced the risk of preeclampsia by 46%.

Researchers have reported a 21% reduction of maternal brain DHA levels during animal gestation when dietary supply of the essential fatty acid is low.  Previous studies had demonstrated that women with lower blood or breast milk DHA content were more likely to suffer from postpartum depression.  It had previously been thought that brain DHA levels did not fluctuate because the nutrient is eagerly preserved. The authors concluded that the high ratio of omega-6 to omega-3 fatty acids in the Western diet might be a risk factor for postpartum depression.

Alcohol inhibits delta-6 and delta-5 desaturases, which suppresses the conversion of linoleic acid and gamma-linolenic acid to arachidonic acid (AA), DHA and EPA.  Ethanol also reduces the blood levels of linoleic acid.  Alcohol consumption by pregnant women therefore reduces the availability of essential fatty acids to the developing fetus and may partially explain the developmental deficits seen in Fetal Alcohol Syndrome.  This also explains why vitamin B3, necessary for delta-6 desaturase activity, exerts neuroprotective activity in fetal alcohol syndrome.


Other Benefits

DHA is also important for cardiovascular health, and can help improve cholesterol balance, protect the blood vessels from damage and regulate blood pressure levels. It also has powerful anti-inflammatory effects, and is an important factor for the maintenance of good health.

 

Purity

Omega Natal™ is a special molecular distillate of fish oil that provides super concentrated amounts of DHA along with EPA. This molecular distillation process allows for the removal of heavy metals such as mercury, organochlorine contaminants such as polychlorinated biphenyls (PCBs), and chemical solvents, making it safe and effective for everyday use during pregnancy. Omega Natal™ is also an uniquely stable fish oil, both because of its pharmaceutical-quality, ultra-low peroxide value and because of the unique blend of synergistic fat soluble antioxidants. Studies show that stable fish oil has superior benefits as compared with common commercial grades oils.

 

References

Fleith M, Clandinin MT. Dietary PUFA for preterm and term infants: review of clinical studies. Crit Rev Food Sci Nutr. 2005;45(3):205-29. Review.

Chong EW, Sinclair AJ, Guymer RH. Facts on fats. Clin Experiment Ophthalmol. 2006 Jul;34(5):464-71.

Clandinin MT, Chappell JE, Leong S, Heim T, Swyer PR, Chance GW. Intrauterine fatty acid accretion rates in human brain: implications for fatty acid requirements. Early Hum Dev. 1980 Jun;4(2):121-9.

Clandinin MT. Brain development and assessing the supply of polyunsaturated fatty acid. Lipids. 1999 Feb;34(2):131-7.

Lanting CI, Fidler V, Huisman M, Touwen BC, Boersma ER. Neurological differences between 9-year-old children fed breast-milk or formula-milk as babies. Lipids. 1999 Feb;34(2):151-60.

Olsen SF, Secher NJ. Low consumption of seafood in early pregnancy as a risk factor for preterm delivery: prospective cohort study. BMJ. 2002 Feb 23;324(7335):447.

Weisinger HS, Armitage JA, Sinclair AJ, Vingrys AJ, Burns PL, Weisinger RS. Perinatal omega-3 fatty acid deficiency affects blood pressure later in life. Nat Med. 2001 Mar; 7(3):258-9.

Troxell H, Anderson J, Auld G, Marx N, Harris M, Reece M, Allen K. Omega-3 for baby and me: material development for a WIC intervention to increase DHA intake during pregnancy. Matern Child Health J. 2005 Jun;9(2):189-97.

Wang YP, Kay HH, Killam AP. Decreased levels of polyunsaturated fatty acids in preeclampsia. Am J Obstet Gynecol. 1991 Mar;164(3):812-8.


Dietary PUFA for preterm and term infants: review of clinical studies

Fleith M, Clandinin MT. Crit Rev Food Sci Nutr. 2005;45(3):205-29. Review.

Human milk contains n-3 and n-6 LCPUFA (long chain polyunsaturated fatty acids), which are absent from many infant formulas. During neonatal life, there is a rapid accretion of AA (arachidonic acid) and DHA (docosahexaenoic acid) in infant brain, DHA in retina and of AA in the whole body. The DHA status of breast-fed infants is higher than that of formula-fed infants when formulas do not contain LCPUFA. Studies report that visual acuity of breast-fed infants is better than that of formula-fed infants, but other studies do not find a difference. Cognitive development of breast-fed infants is generally better, but many sociocultural confounding factors may also contribute to these differences. The effect of dietary LCPUFA on FA status, immune function, visual, cognitive, and motor functions has been evaluated in preterm and term infants. Plasma and RBC FA status of infants fed formulas supplemented with both n-3 and n-6 LCPUFA was closer to the status of breast-fed infants than to that of infants fed formulas containing no LCPUFA. Adding n-3 LCPUFA to preterm-infant formulas led to initial beneficial effects on visual acuity. Few data are available on cognitive function, but it seems that in preterm infants, feeding n-3 LCPUFA improved visual attention and cognitive development compared with infants receiving no LCPUFA. Term infants need an exogenous supply of AA and DHA to achieve similar accretion of fatty acid in plasma and RBC (red blood cell) in comparison to breast-fed infants. Fewer than half of all studies have found beneficial effects of LCPUFA on visual, mental, or psychomotor functions. Improved developmental scores at 18 mo of age have been reported for infants fed both AA and DHA. Growth, body weight, and anthropometrics of preterm and term infants fed formulas providing both n-3 and n-6 LCPUFA fatty acids is similar in most studies to that of infants fed formulas containing no LCPUFA. A larger double-blind multicenter randomized study has recently demonstrated improved growth and developmental scores in a long-term feeding study of preterm infants. Collectively, the body of literature suggests that LCPUFA is important to the growth and development of infants. Thus, for preterm infants we recommend LCPUFA intakes in the range provided by feeding of human milk typical of mothers in Western countries. This range can be achieved by a combination of AA and DHA, providing an AA to DHA ratio of approximately 1.5 and a DHA content of as much as 0.4%. Preterm infants may benefit from slightly higher levels of these fatty acids than term infants. In long-term studies, feeding more than 0.2% DHA and 0.3% AA improved the status of these fatty acids for many weeks after DHA; AA was no longer present in the formula, enabling a DHA and AA status more similar to that of infants fed human milk. The addition of LCPUFA in infant formulas for term infants, with appropriate regard for quantitative and qualitative qualities, is safe and will enable the formula-fed infant to achieve the same blood LCPUFA status as that of the breast-fed infant.

 

 

Intrauterine fatty acid accretion rates in human brain: implications for fatty acid requirements.

Clandinin MT, Chappell JE, Leong S, Heim T, Swyer PR, Chance GW. Early Hum Dev. 1980 Jun;4(2):121-9.

Fatty acid components of infant brain were determined to assess fatty acid requirements for synthesis of structural lipids in brain tissue during the last trimester of development in the fetus. Quantitative fatty acid analysis of cerebellum, frontal and occipital brain lobes indicated rapid accretion of chain elongation and desaturation products during the last trimester of brain growth. Frontal and occipital brain lobes were similar in fatty acid content. Fatty acid accretion rates were determined by regression analysis of tissue fat components at varying gestational ages. Tissue accretion of saturated and omega-9 fatty acids, as well as total fatty acid content, paralleled increases in whole brain weight. Levels of linoleic (C18:2, omega-6) and linolenic (C18:3, omega-3) acids were consistently low in brain during the last trimester of development, while marked substantial accretion of long chain desaturation products, arachidonic (C20:4, omega-6) and docosahexaenoic (C22:6, omega-3) acids occurred. Accretion of individual fatty acids of cerebellum also reflected changes in tissue total fatty acid content, with exception of the levels of C18:3, omega-3 and its chain elongation products present in cerebellum during the last trimester. These developmental changes and estimates of fatty acid incorporation into whole brain and cerebellum are quantitatively relevant to estimation of fatty acid requirements of the low birth weight neonate.

 

Brain development and assessing the supply of polyunsaturated fatty acid.

Clandinin MT. Lipids. 1999 Feb;34(2):131-7.

embrane lipids are necessary for structure and function of the developing nervous system. Rapid synthesis of brain tissue occurs during the last trimester of development of the human brain and the early postnatal weeks. This synthesis of brain structure involves the formation of complex lipids, many of which contain significant quantities of chain-elongated desaturated homologs of essential fatty acids. The present report discusses the implications of change in nutritional status on processes of brain development and metabolic events that involve lipids.

 

Omega-3 for baby and me: material development for a WIC intervention to increase DHA intake during pregnancy.

Troxell H, Anderson J, Auld G, Marx N, Harris M, Reece M, Allen K. Matern Child Health J. 2005 Jun;9(2):189-97.

OBJECTIVE: The purpose of this project was to develop educational materials for a Special Supplemental Nutrition Program for Women, Infants, and Children (WIC) program intervention in Denver, Colorado. Despite accumulating evidence of the importance of docosahexaenoic acid (DHA) during pregnancy, there has been no attempt to develop a targeted nutrition intervention to improve birth outcomes among high-risk women. The goal of the Omega-3 for Baby and Me intervention, for which these materials were developed, is to increase the consumption of DHA-rich foods to decrease the risk for premature delivery and low-birth weight babies among this population. METHOD: Following collection of data from eight focus groups among the target audience, the principles of the Health Belief Model were used to develop materials to address the needs, barriers, and motivators of this audience. In addition, process evaluation pilot testing was used to evaluate recipes and logos during the material development. RESULTS: Materials developed for this intervention include a logo, 9-month calendar, stickers, shopping lists, recipes, recipe holder, magnetic clip, nylon pouch, and recruitment materials. CONCLUSION: The use of focus groups and pilot testing increased the target audience acceptance of education materials that are being used for the Omega-3 for Baby and Me intervention. Successful outcomes from the Omega-3 for Baby and Me intervention, i.e., decreasing the incidence of preterm birth and low-birth weight, will have implications for supplemental food policies at state and national levels.

Decreased levels of polyunsaturated fatty acids in preeclampsia.

Wang YP, Kay HH, Killam AP. Am J Obstet Gynecol. 1991 Mar;164(3):812-8.

Plasma levels of polyunsaturated fatty acids in the n-3 and n-6 classes, which include linoleic, linolenic, arachidonic, eicosapentaenoic, and docosahexaenoic acids, were quantified with high-performance liquid chromatography in nonpregnant volunteers and in patients with normal pregnancies or preeclampsia at term. The total polyunsaturated fatty acid levels were not significantly different between nonpregnant and normal pregnant patients but was significantly lower in the preeclamptic patients compared with normal pregnant patients. This decreased level could represent altered fatty acid metabolism or altered storage and mobilization from lipid pools. Compared with nonpregnant patients, normal pregnant patients had significantly higher levels of eicosapentaenoic and docosahexaenoic acid. This may reflect normal physiologic changes in pregnancy, and the decreased level of eicosapentaenoic acid seen in preeclamptic patients may play a significant role in the pathophysiology of preeclampsia.

 

 






SKU Number: AOR04215
Prenatal
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: AOR04215





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